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GHK-Cu 50mg

GHK-Cu 50mg

$39.00Price

What is GHK-CU?

Specifically, GHK-CU is a naturally occurring copper-peptide complex comprised of three amino acids: glycine, histidine, and lysine. In particular, biochemist Loren Pickart first isolated GHK-CU from human plasma in the 1970s.

How Does GHK-CU Work?

Primarily, GHK-CU activates receptors involved in tissue repair processes. This stimulates cells to increase collagen and elastin production. Additionally, GHK-CU exhibits antioxidant effects by scavenging free radicals. Moreover, it helps shuttle copper ions to cells for biological activity.

What Are the Benefits of GHK-CU?

By stimulating regenerative processes, GHK-CU 100mg can:

  • Accelerate wound healing
  • Improve skin appearance and texture
  • Reduce inflammation and oxidative damage
  • Promote tissue repair and regeneration

Numerous studies show GHK-CU can accelerate wound healing. For example, GHK-CU improved wound closure in rats by over 50% compared to control see study.

Additionally, GHK-CU reduces inflammation caused by UV radiation see study. It also promotes regeneration in animal models of tissue injury see study.

Furthermore, research indicates GHK-CU improves skin appearance by stimulating collagen. In clinical trials, GHK-CU tightened loose skin and improved skin elasticity and firmness see clinical trial results.

 

What is the Technical Data?

Firstly, GHK-CU consists of a short 3 amino acid chain with the sequence glycine-histidine-lysine (GHK).

Additionally, it contains the copper binding amino acid histidine. Moreover, glycine and lysine provide stability.

Furthermore, the molecular formula of GHK-CU is C18H30N6O6Cu and it has a copper ion (Cu2+) bound to the peptide.

Also, the structure of GHK-CU enables the copper ion to form coordination bonds with the histidine amino acid. Consequently, this facilitates shuttling of copper within the body.

Finally, the small tripeptide design of GHK-CU allows for good bioavailability and cell penetration. Thus, GHK-CU 100mg can readily exert its copper-mediated biological effects.

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